College

Buchtel College of Arts and Sciences

Date of Last Revision

2026-05-07 06:09:21

Major

Biomedical Science

Honors Course

BIOL 499

Number of Credits

4

Degree Name

Bachelor of Science

Date of Expected Graduation

Spring 2026

Abstract

Osteoporosis and vertebral degeneration are major contributors to musculoskeletal morbidity, yet the molecular mechanisms regulating vertebral bone and intervertebral disc (IVD) homeostasis remain incompletely understood. GPNMB (osteoactivin) has been identified as a regulator of bone remodeling, though its role in spine health is unclear. This study evaluated the effects of GPNMB deficiency on lumbar vertebral bone microarchitecture and IVD integrity in a sex-dependent context.

Six-month-old male and female wild-type (WT) and GPNMB knockout (KO) mice were analyzed using micro–computed tomography (µCT) of lumbar vertebrae (L4–L6) and histological assessment of the L5–L6 IVD. KO males exhibited increased bone volume fraction, bone surface density, and trabecular number, alongside decreased trabecular thickness and separation, indicating a shift toward a more numerous but thinner trabecular network. KO females showed a milder phenotype, with increased bone surface-to-volume ratio and reduced trabecular thickness. Cortical bone deficits were most pronounced in KO males, while females exhibited greater cortical bone volume overall. Histological analysis revealed increased IVD degeneration in KO males compared to WT males, with no differences observed in females.

These findings suggest that GPNMB plays a critical, sex-dependent role in vertebral bone and disc homeostasis.

Research Sponsor

Brian Bagatto

First Reader

Fayez F. Safadi

Second Reader

Michael DiSabato

Honors Faculty Advisor

Brian Bagatto

Proprietary and/or Confidential Information

No

Community Engaged Scholarship

Yes

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